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Preop when you had conductive hearing loss and H, did you also have a low frequency T? Or only the high-pitched T that remained postop? And just to be clear, no echoing, no thumping. The H was just popping?

So on a single instance of keyboard type you would have a single pop or a series of pops, and it was until the end of the sound or beyond the end of the sound?

Re-reading your account, @GregCA I can't remember if I asked you elsewhere what exactly did your hyperacusis feel like and to what sounds were you sensitive to?

Interesting is just the tip of the iceberg. Both the cochlea theory and the central gain theory make no sense to explain @GregCA recovery, and yet they're mainstream. @GregCA had crackling, I have thumping and vibration, and joe schmoe has a banana in his ear. ''Hyperacusis'' is the bastard son...

So conductive HL goes, sensorineural HL remains, and yet we're supposed to believe the circus of hyperacusis conference muppets claiming the fault lies in sensorineural hidden hearing loss and synaptopathy. Btw, I'm quite sure their claim used to be SNHL without being hidden at all, because...

What else could it be if I'm seeing people say things like ''the middle ear can lose it's air cushion effect and make the tympanic membrane vibrate more than it should''. That makes so much more sense than synaptopathy hidden hearing loss and glial thingies.

Most doctors believe in central gain and that the middle ear has nothing to do with H, and will say that was only because you recovered the hearing that you'd temporarily lost before the op. I think you answered me this the other time we spoke, but was your H improvement a lot larger than your...

Well I have enough on my table and don't know what recruitment is. I read Bungler's , Bauman's and then @AnxiousJon said his understanding of what Harold Kim told him was that the tensor tympani was the one involved in making certain frequencies seem amplified, that if the dysacusia were only...

Isn't recruitment like a feature of ''meniere's'' that thing ENTs don't agree what it actually is? Well an austrian guy named Franz claims to deal with menieres through tenotomy of the middle ear muscles, just saying... he says something along the lines ''enlarged cochlear aqueduct--- middle ear...

Harold Kim reportedly told someone on the forum that the middle ear was capable of imitating external sounds (dysacusis?), but apparently everyone in the West USA prefers to read the NY Post and tinnitustalk instead of verifying how deep these claims go.

I doubt that the auditory nerve also pass through the eustachian tube and those patients with both hyperacusis and autophonia from issues in the tube. What about that patient with hyperacusis over at the facebook Tonic tensor tympani support group who was diagnosed by germany's PET specialist as...

Hyperacusis focus mentions the middle ear as a potential mechanism. None of those writers on the cochlea you like so much write about whether the pain fibers they've supposedly discovered could just be pain fibers in the middle ear. Hell, in the TTTS support group on fb we have one that could be...

There's massive overlap in diagnosis in mainstream otology, there's obviously gonna be even more overlap in an area otologists don't give a rat's ass about. Jastreboff flew in to take advantage of this lack of interest, and now the cochlear damage bunch are doing the same. This site has a post...

You have no proof of what you say. Acoustic trauma has been linked to straight up myoclonus as well as TTTS as well as ''hyperacusis''. And now in the article we have an even bigger range of symptoms on a TMJ issue, which has long been linked to TTTS a lot more than it's been linked to...

This may raise awareness, but it just shows what a ridiculous concept hyperacusis is. Those sxs are most probably tonic tensor tympani syndrome or veli palatini muscle fibers or eustachian tube dysfunction. Not a missing link between cochlear hearing loss and ''hyperacusis'' based on some...