https://www.ata.org/news/news/hyperacusis-related-damage-nerve-cells-inner-ear Research from John Hopkins University said most likely has something to do with a minority of outer hair cell nerve fibers acting as pain receptors.
-
Don't miss the opportunity to attend Tinnitus Quest's online Q&A with Dr. Susan Shore and Jon Pearson, CEO of Auricle on October 13. We will hear the latest about the Susan Shore Device.
➡️ Details & Registration!
Tinnitus Talk Support Forum
This page states hyperacusis is triggered by OHC's which release ATP which activates the Type II afferent nerve fibers to act as pain receptors. This happens naturally with 120db+ noise but the hypothesis is painful hyperacusis lowers the tolerance level that activates the Type II afferent nerve fibers once OHC's are damaged.