The complete title is "Pinpointing a Highly Specific Pathological Functional Connection That Turns Phantom Sound into Distress.". Again i have access to the abstract only. But some sentences are interesting. By De Ridder, Congedo and Vanneste
Electroencephalography data of 317 tinnitus patients and 256 healthy subjects were analyzed, using independent component analysis
So i guess it is not underpowered.
Results demonstrate that tinnitus is characterized by at least 2 major brain networks, each consisting of multiple independent components. One network reflects tinnitus distress, while another network reflects the loudness of the tinnitus.
Self explanatory
The distress and loudness networks do not intercommunicate for patients without distress, but do when patients are distressed by their tinnitus.
That being said, they did not say that distress and loudness are well correlated. Just that the neural networks communicate. Naive question: for the patients who have no distress, what is their distress network that does not communicate ?
http://www.ncbi.nlm.nih.gov/pubmed/23632885
Electroencephalography data of 317 tinnitus patients and 256 healthy subjects were analyzed, using independent component analysis
So i guess it is not underpowered.
Results demonstrate that tinnitus is characterized by at least 2 major brain networks, each consisting of multiple independent components. One network reflects tinnitus distress, while another network reflects the loudness of the tinnitus.
Self explanatory
The distress and loudness networks do not intercommunicate for patients without distress, but do when patients are distressed by their tinnitus.
That being said, they did not say that distress and loudness are well correlated. Just that the neural networks communicate. Naive question: for the patients who have no distress, what is their distress network that does not communicate ?
http://www.ncbi.nlm.nih.gov/pubmed/23632885