So, this one is kind of interesting: http://en.wikipedia.org/wiki/Fasoracetam http://nootriment.com/fasoracetam/ GABAb is strongly implicated as a tinnitus vector in a couple studies. http://www.ncbi.nlm.nih.gov/pubmed/15570498 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3360237/ The (completely unavailable) l-isomer of baclofen in isolation from the d-isomer that's included with it in all commercial baclofen products, had a strong effect on tinnitus: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3297816/ So, then the question is "if a lack of GABAb initiated inhibition is involved in tinnitus signal propagation, and if Fasoracetam can specifically upregulate GABAb receptors, will consumption of fascoracetam decrease tinnitus signal strength?" I think it is an interesting question, enough so that I acquired samples of fasoracetam from a couple different vendors. Unfortunately, this stuff doesn't even have "supplement" status, meaning that any you are likely to acquire will be made by a chinese lab and come in a mailer that says "RESEARCH SAMPLE. NOT FOR HUMAN CONSUMPTION". My days of willingly consuming such things are probably a little bit behind me. But, it is out there! I will be keeping my eyes open for a USP grade source of this material.